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Nocturnal hypertension and cardiovascular risk: consequences for diagnosis and treatment

L Grote1, J Mayer, T Penzel

  • 1Sleep Disorders Centre, University of Marburg, Germany.

Insights

New blood pressure guidelines identify "nondippers" at higher cardiovascular risk. Addressing sleep-related breathing disorders and nocturnal hypertension is crucial for effective treatment and improved patient outcomes.

Area of Science:

  • Cardiology
  • Sleep Medicine
  • Pharmacology

Background:

  • Ambulatory 24-hour blood pressure monitoring reveals distinct circadian patterns, classifying individuals as
  • dippers
  • or
  • nondippers
  • (lacking a normal nighttime blood pressure drop).
  • Nondipping status is increasingly linked to essential hypertension and elevated risk of cardiovascular complications, including myocardial infarction and cerebrovascular events.
  • Sleep-related breathing disorders (SRBD) are associated with increased cardiovascular risk, potentially causing significant blood pressure elevations during REM sleep.

Purpose of the Study:

  • To investigate the diagnostic and therapeutic implications of altered circadian blood pressure profiles, particularly in nondippers.
  • To evaluate the impact of sleep-related breathing disorders on 24-hour blood pressure patterns and cardiovascular risk.
  • To assess the efficacy of antihypertensive therapy in managing both daytime and nighttime blood pressure, including nocturnal hypertension.

Main Methods:

  • Utilized ambulatory noninvasive 24-hour blood pressure monitoring to classify patients into dipper and nondipper categories.
  • Examined the correlation between nondipping patterns, sleep-related breathing disorders (SRBD), and cardiovascular risk factors in a general practice population.
  • Employed a new protocol for 24-hour blood pressure assessment under standardized load, incorporating nocturnal hypertension evaluation.

Main Results:

  • A high incidence (40/112) of nondippers was observed in a group of snoring, obese middle-aged men with daytime fatigue, suggesting a link to SRBD.
  • Antihypertensive drug therapy aims to reduce blood pressure during both day and night, across wakefulness and sleep stages.
  • The angiotensin-converting enzyme (ACE) inhibitor cilazapril demonstrated a significant 24-hour mean blood pressure reduction of 10.0 mm Hg compared to placebo (4.3 mm Hg).

Conclusions:

  • Altered circadian blood pressure profiles, especially in nondippers, necessitate consideration of nocturnal breathing patterns and daytime symptoms related to SRBD.
  • Effective antihypertensive strategies must address 24-hour blood pressure control, including nocturnal hypertension.
  • Cilazapril shows promise in managing hypertension by significantly reducing 24-hour blood pressure, offering a potential therapeutic option for nondippers and those with SRBD.

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