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R-plasmid transfer in vivo in the absence of antibiotic selection pressure

Insights

An O18 Escherichia coli strain carrying an R plasmid became dominant after tetracycline therapy. Later, an O88 E. coli strain with a similar R plasmid appeared, suggesting in-vivo transfer without antibiotics.

Area of Science:

  • Microbiology
  • Genetics
  • Epidemiology

Background:

  • Antibiotic resistance is a growing public health concern.
  • R plasmids mediate resistance in bacteria like Escherichia coli.
  • Understanding R plasmid dynamics in the gut microbiome is crucial.

Purpose of the Study:

  • To track the persistence and transfer of an R plasmid within an individual's fecal flora.
  • To investigate the potential for R plasmid transfer between Escherichia coli strains in vivo.
  • To characterize the molecular similarity of R plasmids in different E. coli strains.

Main Methods:

  • Longitudinal fecal sampling over 9 months.
  • Detection and isolation of specific Escherichia coli strains (O18 and O88).
  • Molecular characterization of isolated R plasmids.

Main Results:

  • An O18 E. coli strain with an R plasmid achieved dominance after tetracycline treatment.
  • No R plasmid transfer was detected for 202 days post-treatment.
  • An O88 E. coli strain with a similar R plasmid emerged, indicating potential in-vivo transfer.
  • Molecular analysis confirmed similarity between the R plasmids from O18 and O88 strains.

Conclusions:

  • R plasmid transfer between E. coli strains can occur in the human gut without ongoing antibiotic pressure.
  • The human gut microbiome can facilitate horizontal gene transfer of antibiotic resistance.
  • This study highlights the complex dynamics of R plasmid persistence and dissemination in vivo.

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