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Experience with Enterobacter bacteraemia in a Dublin teaching hospital
N al Ansari1, E B McNamara, R J Cunney
1Department of Microbiology, Beaumont Hospital, Dublin, Ireland.
The Journal of Hospital Infection
|May 1, 1994
Summary
Enterobacter bacteraemia is linked to central venous catheters and H-2 receptor antagonists. These factors, plus prior gentamicin exposure and ICU stay, increase gentamicin-resistant Enterobacter infections.
Area of Science:
- Infectious Diseases
- Hospital Epidemiology
- Antimicrobial Resistance
Background:
- Enterobacter bacteraemia is a significant hospital-acquired infection.
- Risk factors for Enterobacter infections require further investigation.
- Understanding resistance patterns is crucial for effective treatment.
Purpose of the Study:
- To identify risk factors associated with Enterobacter bacteraemia.
- To determine factors contributing to gentamicin-resistant Enterobacter bacteraemia.
- To inform empirical antibiotic strategies for Gram-negative septicaemia.
Main Methods:
- Retrospective review of Enterobacter bacteraemia episodes over a 2-year period.
- Analysis of patient data to identify potential risk factors.
- Evaluation of associations between risk factors and infection outcomes.
Main Results:
- Twenty-two episodes of Enterobacter bacteraemia were identified in 20 patients.
- Central venous catheters and H-2 receptor antagonist use were associated with Enterobacter infection.
- Gentamicin resistance was linked to prior gentamicin exposure and prolonged intensive care unit (ICU) stay.
Conclusions:
- Central venous catheters and H-2 receptor antagonists are risk factors for Enterobacter bacteraemia.
- Previous gentamicin exposure and ICU stay increase the risk of gentamicin-resistant strains.
- Findings impact empirical antibiotic choices for suspected Gram-negative septicaemia.