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P2Z adenosine triphosphate receptor activity in cultured human monocyte-derived macrophages

S E Hickman1, J el Khoury, S Greenberg

  • 1Department of Physiology and Cellular Biophysics, Columbia University, College of Physicians and Surgeons, New York, NY 10032.

Blood
|October 15, 1994
PubMed

Insights

Human mononuclear phagocytes have a P2Z-like receptor. Extracellular adenosine triphosphate (ATP) creates pores, allowing dye entry, with increased activity in macrophages and distinct properties from mouse cells.

Area of Science:

  • Immunology
  • Cell Biology
  • Biochemistry

Background:

  • Mononuclear phagocytes play crucial roles in immunity and inflammation.
  • Purinergic signaling via P2 receptors is involved in various cellular processes.
  • Understanding purinergic receptor subtypes and their functions is essential for therapeutic development.

Purpose of the Study:

  • To investigate the presence and characteristics of P2Z-like purinergic receptor activity in human mononuclear phagocytes.
  • To compare the pharmacological properties of human P2Z-like receptors with their murine counterparts.
  • To examine the developmental regulation of P2Z-like receptor expression during monocyte-to-macrophage differentiation.

Main Methods:

  • Human mononuclear phagocytes (monocytes and macrophages) were utilized.
  • Extracellular adenosine triphosphate (ATP) was used to induce membrane permeabilization.
  • Fluorescent dyes (YO-PRO-1, Lucifer yellow) were employed to assess membrane integrity.
  • Pharmacological agents including Mg2+, oxidized ATP, Benzoylbenzoic-ATP (BzBzATP), and adenosine-5 -O-(thiophosphate) (ATPγS) were used to characterize receptor activity.
  • Murine macrophage-like J774 cells were used for comparative studies.

Main Results:

  • Human mononuclear phagocytes express a P2Z-like purinergic receptor activity.
  • Extracellular ATP induced nonselective membrane pore formation, allowing dye entry.
  • ATP-induced permeabilization increased with monocyte maturation into macrophages.
  • Receptor activity was inhibited by Mg2+ and oxidized ATP.
  • BzBzATP and ATPγS showed differential efficacy in human macrophages compared to ATP, unlike in murine J774 cells.
  • UTP, GTP, and CTP were ineffective in permeabilizing either human or murine macrophages.

Conclusions:

  • Human monocyte-derived macrophages express a P2Z-like purinergic receptor activity.
  • This human receptor exhibits distinct pharmacological properties compared to the murine P2Z receptor.
  • The expression of these P2Z-like receptors is developmentally regulated during human mononuclear phagocyte differentiation.

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