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Morphine-isoflurane interaction in dogs, swine and rhesus monkeys

E P Steffey1, J D Baggot, J H Eisele

  • 1Department of Surgery, School of Veterinary Medicine, University of California, Davis 95616.

Insights

Morphine significantly reduces the minimum alveolar concentration (MAC) of isoflurane anesthesia in monkeys, dogs, and swine. It also causes a sustained increase in PaCO2, which is reversed by naloxone.

Area of Science:

  • Veterinary Anesthesiology
  • Pharmacology

Background:

  • Isoflurane is a common inhalation anesthetic.
  • Morphine is an opioid analgesic with known anesthetic-sparing effects.
  • Understanding species-specific responses to anesthetic combinations is crucial for safe practice.

Purpose of the Study:

  • To evaluate the anesthetic-sparing effect of morphine on isoflurane MAC in different species.
  • To investigate the impact of morphine administration on PaCO2 during spontaneous ventilation.
  • To assess the reversibility of morphine-induced respiratory effects with naloxone.

Main Methods:

  • Three species (monkeys, dogs, swine) were administered intravenous morphine (2 mg/kg).
  • The reduction in isoflurane MAC and changes in PaCO2 were monitored.
  • Pharmacokinetic analysis and naloxone reversal were performed.

Main Results:

  • Morphine reduced isoflurane MAC by a minimum of 55% in monkeys, 50% in dogs, and 13% in swine.
  • PaCO2 increased to 55-60 mmHg with morphine and remained elevated despite falling plasma concentrations.
  • Naloxone administration promptly reversed the elevated PaCO2.

Conclusions:

  • Morphine exhibits significant anesthetic-sparing effects on isoflurane across species, with varying potency.
  • Morphine causes a persistent hypercapnic effect independent of plasma concentration, suggesting central respiratory depression.
  • Naloxone effectively antagonizes morphine-induced hypercapnia, highlighting opioid receptor involvement.

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