Neurotransmitter release from viable purely cholinergic Torpedo synaptosomes

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Apolipoprotein E4 decreases whereas apolipoprotein E3 increases the level of secreted amyloid precursor protein after closed head injury.

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Lack of synucleins induces an alteration of lipid peroxidation in the brain.

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Single-cell reanalysis characterizes an Osmr+ astrocyte state and predicts midkine signaling to Cox6b1+ glutamatergic neurons at 24 h after traumatic brain injury.

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Related Experiment Videos

Neurotransmitter release from viable purely cholinergic Torpedo synaptosomes

Related Concept Videos

The Interplay Between Apolipoprotein E4 and the Autophagic-Endocytic-Lysosomal Axis.

Opposing actions of environmental enrichment and Alzheimer's disease on the expression of hippocampal microRNAs in mouse models.

Lack of apolipoprotein-E exacerbates experimental allergic encephalomyelitis.

Apolipoprotein E4 decreases whereas apolipoprotein E3 increases the level of secreted amyloid precursor protein after closed head injury.

Involvement of nuclear factor-kappaB in endothelin-A-receptor-induced proliferation and inhibition of apoptosis.

The effects of APOE genotype on age at onset and progression of neurodegenerative diseases.

Structure-informed characterization of a TLR2-binding scFv reveals a CDR-centered recognition model.

Identification of molecular subtypes in clear cell renal cell carcinoma based on chromatin regulators and tumor immune microenvironment profiling.

Nicotinamide promotes lipophagy and cholesterol efflux through the SIRT1-LXRα deacetylation-ABCA1 pathway in vascular smooth muscle cells.

Lack of synucleins induces an alteration of lipid peroxidation in the brain.

USP33-mediated deubiquitination stabilizes YTHDF2 and promotes glioblastoma malignancy and temozolomide resistance.

Structural basis for the inhibition of HKU8 main protease by nirmatrelvir and ensitrelvir.

Single-cell reanalysis characterizes an Osmr+ astrocyte state and predicts midkine signaling to Cox6b1+ glutamatergic neurons at 24 h after traumatic brain injury.

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Neurotransmitter release from viable purely cholinergic Torpedo synaptosomes

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