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Spermine-induced precocious intestinal maturation in suckling rats: possible involvement of glucocorticoids

M Kaouass1, J Sulon, P Deloyer

  • 1Laboratory of Biochemistry and General Physiology, Institute of Chemistry, Liège, Belgium.

Insights

Spermine accelerates intestinal maturation in young rats by stimulating the pituitary-adrenal axis. This polyamine likely triggers gastrointestinal hormones, indirectly boosting ACTH and corticosterone levels for enhanced gut development.

Area of Science:

  • Endocrinology
  • Gastroenterology
  • Developmental Biology

Background:

  • Polyamines, such as spermine, play crucial roles in cellular processes.
  • Precocious maturation of the gastrointestinal tract is essential for nutrient absorption and development.
  • The hormonal regulation of early intestinal development is not fully understood.

Purpose of the Study:

  • To investigate the mechanisms underlying spermine-induced precocious intestinal maturation in unweaned rats.
  • To determine the role of the hypophysial-adrenal axis in this developmental process.
  • To explore the potential involvement of gastrointestinal hormones in mediating spermine's effects.

Main Methods:

  • Oral administration of spermine to 11-day-old rats.
  • Measurement of serum ACTH and corticosterone levels at various time points.
  • Intraperitoneal administration of spermine and various gastrointestinal hormones.
  • Assay of intestinal disaccharidase activity.

Main Results:

  • Oral spermine significantly increased ACTH and corticosterone secretion in a time-dependent manner.
  • Peak levels of ACTH and corticosterone were observed 4-6 hours post-spermine ingestion.
  • Intraperitoneal spermine did not affect corticosterone levels, and tested gastrointestinal hormones did not alter disaccharidase activity.

Conclusions:

  • The hypophysial-adrenal axis is implicated in spermine-induced postnatal intestinal development.
  • Spermine likely stimulates ACTH and corticosterone secretion indirectly, possibly via gastrointestinal hormone release.
  • These findings suggest a novel hormonal pathway regulating early intestinal maturation.

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