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Three different cDNAs encoding mouse D-factor/LIF receptor

M Tomida1, Y Yamamoto-Yamaguchi, M Hozumi

  • 1Department of Chemotherapy, Saitama Cancer Center Research Institute, Inc.

Insights

Researchers isolated mouse differentiation-stimulating factor (D-factor)/leukemia inhibitory factor (LIF) receptor cDNAs, identifying a soluble form. This soluble receptor, abundant in the liver, is produced via alternative mRNA processing.

Area of Science:

  • Molecular Biology
  • Genetics
  • Biochemistry

Background:

  • Differentiation-stimulating factor (D-factor), also known as leukemia inhibitory factor (LIF), plays a crucial role in cellular differentiation.
  • Understanding the receptor for D-factor/LIF is essential for elucidating its biological functions.

Purpose of the Study:

  • To isolate and characterize cDNAs encoding the mouse differentiation-stimulating factor (D-factor)/leukemia inhibitory factor (LIF) receptor.
  • To identify potential isoforms of the D-factor/LIF receptor, including soluble forms.

Main Methods:

  • Isolation of mouse liver cDNAs using a probe derived from human sequences.
  • RT-PCR (Reverse Transcription Polymerase Chain Reaction) for probe preparation.
  • Sequence analysis to identify receptor domains and alternative splicing events.

Main Results:

  • Three cDNAs for the mouse D-factor/LIF receptor were isolated, showing high homology to the human counterpart.
  • A soluble receptor isoform was identified, resulting from a 501 bp insertion that introduces a premature stop codon.
  • Alternative polyadenylation of the soluble receptor mRNA leads to two distinct transcript sizes (2.6-3 kb and >5 kb), with shorter transcripts being highly abundant in the liver.

Conclusions:

  • The mouse D-factor/LIF receptor exists in at least two forms: a full-length cellular receptor and a soluble receptor.
  • The soluble D-factor/LIF receptor is generated through alternative splicing and exhibits differential mRNA processing, with abundant expression in the liver.
  • These findings provide insights into the molecular mechanisms regulating D-factor/LIF signaling and receptor availability.

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