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Biochemical and functional characterization of aminopeptidase N expressed by human melanoma cells

A Menrad1, D Speicher, J Wacker

  • 1Wistar Institute of Anatomy and Biology, Philadelphia, Pennsylvania 19104.

Cancer Research
|March 15, 1993
PubMed

Insights

Aminopeptidase N, a protein found on melanoma cells, aids in cancer invasion. Inhibiting this enzyme blocks melanoma cells from penetrating basement membranes, suggesting it is a potential therapeutic target.

Area of Science:

  • Biochemistry
  • Molecular Biology
  • Oncology

Background:

  • Melanoma cells express unique cell surface proteins not found on normal melanocytes.
  • Understanding these proteins is crucial for identifying melanoma-specific targets.
  • Aminopeptidase N (APN) is a cell surface enzyme implicated in various cellular processes.

Purpose of the Study:

  • To biochemically and functionally characterize a cell surface protein on melanoma cells.
  • To investigate the role of aminopeptidase N (APN) in melanoma cell invasion.
  • To determine if APN is associated with extracellular matrix components and contributes to basement membrane degradation.

Main Methods:

  • Affinity purification and microsequencing to identify the cell surface protein.
  • In situ expression, indirect immunofluorescence, and Western blotting to assess APN localization.
  • In vitro invasion assays using a reconstituted basement membrane (Matrigel).
  • Enzyme activity assays to assess APN's ability to degrade extracellular matrix components.

Main Results:

  • The identified protein was confirmed to be aminopeptidase N (APN).
  • APN was found to be tightly associated with extracellular matrix components.
  • Inhibition of APN activity with specific antiserum or competitive inhibitors significantly reduced melanoma cell invasion through Matrigel.
  • Purified APN degraded a M(r) 160,000 protein within Matrigel.

Conclusions:

  • Aminopeptidase N (APN) is expressed on melanoma cells and plays a significant role in their invasive capabilities.
  • APN's association with the extracellular matrix and its enzymatic activity facilitate melanoma cell invasion of basement membranes.
  • Targeting APN may represent a novel therapeutic strategy for inhibiting melanoma metastasis.

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