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[Lateral thalamic infarction. 22 cases]

L Milandre1, C Brosset, R Khalil

  • 1Service de Neurologie, CHU Timone, Marseille.

Presse Medicale (Paris, France : 1983)
|November 27, 1993
PubMed
Summary
This summary is machine-generated.

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Isolated lateral thalamic infarcts are rare, often linked to hypertensive arteriolopathy. This study found these infarcts frequently cause sensory issues and thalamic pain, suggesting arteriolopathy as a primary cause.

Area of Science:

  • Neurology
  • Neuroimaging
  • Vascular Neurology

Context:

  • Isolated lateral thalamic infarcts (LThl) are uncommon cerebrovascular events.
  • Their etiology is often attributed to hypertensive arteriolopathy.
  • Understanding LThl is crucial for diagnosing and managing stroke subtypes.

Purpose:

  • To investigate the clinical characteristics, etiology, and outcomes of isolated lateral thalamic infarcts.
  • To verify the proposed link between hypertensive arteriolopathy and LThl.
  • To analyze neuroimaging findings and associated symptoms in LThl patients.

Summary:

  • A review of 639 cerebral infarcts identified 22 cases of isolated LThl.
  • Patients presented with prominent sensory or motor symptoms, with pulvinar and ventral posterior thalamic nucleus involvement.

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  • Hypertensive arteriolopathy was suggested as the primary cause due to lesion characteristics and patient comorbidities, though artery-to-artery microembolism cannot be excluded.

    • Impact:

    • This study highlights that isolated LThl, while rare, can lead to significant sensory disturbances and chronic thalamic pain.
    • Findings support hypertensive arteriolopathy as a major etiological factor in LThl.
    • The research contributes to a better understanding of thalamic stroke syndromes and their management.