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Mutation of a mutL homolog in hereditary colon cancer

N Papadopoulos1, N C Nicolaides, Y F Wei

  • 1Johns Hopkins Oncology Center, Baltimore, MD 21231.

Science (New York, N.Y.)
|March 18, 1994
PubMed

Insights

Alterations in mismatch repair genes, like hMLH1, cause hereditary nonpolyposis colorectal cancer (HNPCC). This study identified new human mismatch repair genes, including hMLH1, linking them to HNPCC susceptibility.

Area of Science:

  • Genetics
  • Molecular Biology
  • Oncology

Background:

  • Hereditary nonpolyposis colorectal cancer (HNPCC) is linked to DNA mismatch repair gene defects.
  • Previous research identified mutS-related genes involved in some HNPCC cases.

Purpose of the Study:

  • To identify additional human mismatch repair genes.
  • To investigate the role of these genes in hereditary nonpolyposis colorectal cancer.

Main Methods:

  • Searched a large expressed sequence tag database for human mismatch repair genes.
  • Utilized genetic linkage analysis to map identified genes.
  • Screened for mutations in candidate genes within HNPCC families.

Main Results:

  • Discovered three new human mismatch repair genes related to bacterial mutL.
  • Localized one gene, hMLH1, to chromosome 3p21, near known HNPCC susceptibility markers.
  • Identified disease-causing mutations in hMLH1 in HNPCC kindreds.

Conclusions:

  • The hMLH1 gene is a significant cause of hereditary nonpolyposis colorectal cancer.
  • Defects in multiple mismatch repair genes can lead to HNPCC.
  • This expands the understanding of genetic pathways in colorectal cancer development.

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