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Persistent increases in dopamine D2 receptor mRNA expression in basal ganglia following kindling

H A Gelbard1, C D Applegate

  • 1Department of Neurology, University of Rochester Medical Center, NY 14642.

Epilepsy Research
|January 1, 1994
PubMed

Insights

Amygdala kindling significantly increases D2 receptor mRNA in the brain

Area of Science:

  • Neuroscience
  • Molecular Biology
  • Neurochemistry

Background:

  • Epilepsy and seizure disorders are complex neurological conditions.
  • Dopamine receptors, particularly D2 receptors, play a crucial role in brain function.
  • The kindling model is used to study the development of epilepsy.

Purpose of the Study:

  • To investigate the long-term effects of amygdala kindling on D2 receptor mRNA expression.
  • To examine the impact of combined amygdala and piriform cortex kindling on D2 receptor mRNA levels.

Main Methods:

  • Amygdala kindling model in rodents.
  • Densitometric analysis of tissue sections using oligonucleotide probes for D2 receptor cDNA.
  • Quantification of messenger RNA (mRNA) levels in specific brain regions.

Main Results:

  • Amygdala kindling led to a 20-35% increase in D2 receptor mRNA in the nucleus accumbens and striatum.
  • Overkindling (amygdala followed by piriform cortex) resulted in a 150% increase in the nucleus accumbens and 120% in the striatum.
  • No significant hemispheric asymmetries in D2 receptor mRNA were observed.

Conclusions:

  • The kindling process induces a lasting upregulation of extrapyramidal D2 receptor mRNA.
  • These enduring changes in D2 receptor expression may be linked to alterations in seizure susceptibility and behavior.

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