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Activation of a recombinase-deficient mutant recA protein with alternate nucleoside triphosphate cofactors

Y S Meah1, F R Bryant

  • 1Department of Biochemistry, Johns Hopkins University, School of Hygiene and Public Health, Baltimore, Maryland 21205.

Insights

Mutant recA proteins show pH-dependent strand exchange activity, activated when ATP

Area of Science:

  • Molecular Biology
  • Biochemistry
  • Protein Engineering

Background:

  • RecA protein is crucial for DNA repair and recombination.
  • Mutant recA proteins (G160N)recA and (H163A)recA exhibit altered functionality.
  • Standard conditions (pH 7.5) inhibit strand exchange for these mutants.

Purpose of the Study:

  • Investigate the pH-dependent activation of mutant recA proteins in strand exchange.
  • Determine the role of ATP's S0.5 value in recA filament stabilization.
  • Explore alternative cofactor effects on mutant recA activity.

Main Methods:

  • Enzyme kinetics assays measuring ATP hydrolysis and strand exchange.
  • pH variation studies to assess protein activity.
  • Cofactor substitution experiments using dATP and ddATP.

Main Results:

  • Strand exchange activity for (G160N)recA and (H163A)recA is restored at pH 6.0-6.8.
  • Activation correlates with a decrease in S0.5(ATP) below 100 microM for (H163A)recA.
  • (H163A)recA regains strand exchange activity across pH 6.0-8.2 with dATP/ddATP, which have low S0.5 values.

Conclusions:

  • The S0.5 value of nucleoside triphosphates is critical for stabilizing the active recA-DNA filament.
  • pH influences recA activity by modulating the cofactor's S0.5 value.
  • Understanding these parameters aids in designing or understanding recA-like functions.

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