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Isolation of the cyclosporin-sensitive T cell transcription factor NFATp

P G McCaffrey1, C Luo, T K Kerppola

  • 1Division of Tumor Virology, Dana-Farber Cancer Institute, Boston, MA.

Science (New York, N.Y.)
|October 29, 1993
PubMed

Insights

Researchers purified Nuclear Factor of Activated T cells p (NFATp) from T cells and cloned its DNA. This transcription factor is crucial for initiating immune responses by regulating interleukin-2 (IL-2) gene expression.

Area of Science:

  • Immunology
  • Molecular Biology
  • Transcription Factor Research

Background:

  • Nuclear factor of activated T cells (NFAT) regulates interleukin-2 (IL-2) expression in T cells.
  • NFATp, a phosphoprotein, dictates DNA-binding specificity and is targeted by immunosuppressants like cyclosporin A and FK506.

Purpose of the Study:

  • To purify NFATp from murine T cells.
  • To isolate a complementary DNA (cDNA) clone encoding NFATp.
  • To analyze the function of NFATp in T cell transcription.

Main Methods:

  • Purification of NFATp from murine T cells.
  • Isolation of NFATp-encoding cDNA.
  • Expression of a truncated NFATp as a recombinant protein in bacteria.
  • Electrophoretic mobility shift assays (EMSAs) to assess DNA-binding specificity.

Main Results:

  • NFATp was successfully purified from murine T cells.
  • A cDNA clone encoding NFATp was isolated.
  • Recombinant truncated NFATp specifically bound to the NFAT site on the murine IL-2 promoter.
  • Recombinant NFATp formed a transcriptionally active complex.

Conclusions:

  • The molecular cloning of NFATp enables detailed analysis of this key T cell transcription factor.
  • NFATp plays a central role in initiating the immune response.
  • Understanding NFATp function is critical for T cell activation and regulation.

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