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Updated: Aug 15, 2026

Hemodynamic Precision in the Neonatal Intensive Care Unit using Targeted Neonatal Echocardiography
Published on: January 27, 2023
[Ventricular tachycardia of the infant. 2 new cases]
Insights
Infantile ventricular tachycardia (VT) is rapid and irregular, often requiring immediate electric shock treatment. Effective prevention involves procainamide and beta-blockers, with surgical intervention considered for resistant cases.
Area of Science:
- Pediatric Cardiology
- Cardiac Electrophysiology
Background:
- Ventricular tachycardia (VT) in infants is rare but potentially life-threatening.
- Diagnosis can be challenging due to the commonality of pre-excitation syndromes in this age group.
Observation:
- Reported are two new cases of infantile VT, alongside a review of 23 previously documented cases.
- Infantile VTs are characterized by rapid, irregular rhythms with diverse presentations.
- Urgent treatment often involves electrical cardioversion.
Findings:
- Preventive therapy typically combines procainamide and beta-blockers at high doses.
- Diagnostic workup should exclude structural heart abnormalities like cardiomyopathy, tumors, and congenital defects.
- Regular electrical testing and dose adjustments are crucial for long-term management.
Implications:
- Prompt diagnosis and aggressive management are vital for improving outcomes in infantile VT.
- Surgical intervention, such as diathermizing the ectopic focus, may be considered for refractory cases.
- Further research into the etiology and optimal treatment strategies for infantile VT is warranted.
Abstract:
Two new cases of ventricular tachycardia (VT) in the infant are reported, and reviewed in the light of the 23 case histories found in the literature. The diagnosis rests upon eliminating a pre-excitation syndrome, which is so common in this age group. The VTs found in infants are rapid, irregular, and take many different forms. They often necessitate urgent treatment with electric shocks. Preventive treatment consists of a combination of procainamide and beta-blockers in relatively large doses. The search for an aetiological agent should include a haemodynamic and angiocardiographic study of all the chambers of the heart to exclude cardiomyopathy, tumours, papyraceous right ventricle and congenital heart defects. Where no cause can be demonstrated, preventive treatment should be given, with regular electrical testing and other follow-up investigations. An attempt to reduce the drug dosage should be made every 6 months, in hospital. In cases which prove resistant despite adequate treatment, it seems justifiable to carry out a pericardial exploration with the aim of diathermising the ectopic focus; this approach is suggested because of the poor natural history of this type of case.
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