Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Experiment Videos

[Hypercoagulation states]

E Tomoriová1, I Tkác, M Takác

  • 1IV. interná klinika LF UPJS a FN Kosice.

Vnitrni Lekarstvi
|November 1, 1993
PubMed
Summary
This summary is machine-generated.

Hypercoagulability, or increased blood clotting, can stem from inherited or acquired factors. Understanding these causes is crucial for preventing arterial and venous thrombosis.

Related Experiment Videos

Related Concept Videos

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

An association between rs7635818 polymorphism located on chromosome 3p12.3 and the presence of abdominal aortic aneurysm.

Physiological research·2021
Same author

Variants in Pharmacokinetic Transporters and Glycemic Response to Metformin: A Metgen Meta-Analysis.

Clinical pharmacology and therapeutics·2016
Same author

A missense variant in GLP1R gene is associated with the glycaemic response to treatment with gliptins.

Diabetes, obesity & metabolism·2016
Same author

Circulatory and adipose tissue leptin and adiponectin in relationship to resting energy expenditure in patients with chronic obstructive pulmonary disease.

Physiological research·2012
Same author

Pharmacogenomic association between a variant in SLC47A1 gene and therapeutic response to metformin in type 2 diabetes.

Diabetes, obesity & metabolism·2012
Same author

Effects of hypoxia on adipose tissue expression of NFκB, IκBα, IKKγ and IKAP in patients with chronic obstructive pulmonary disease.

Cell biochemistry and biophysics·2012
Same journal

Pharmacological profile of mepolizumab.

Vnitrni lekarstvi·2023
Same journal

Atypical form of Goodpasture's disease.

Vnitrni lekarstvi·2023
Same journal

Differentiated thyroid cancer - possible risks of treatment, suppressive therapy and adherence to current recommendations.

Vnitrni lekarstvi·2023
Same journal

Dyslipidemia - the known unknown.

Vnitrni lekarstvi·2023
Same journal

Internal comorbidities and complications of multiple sclerosis therapy - don't be caught off guard!

Vnitrni lekarstvi·2023
Same journal

Implications of highly suppressive treatment HIV infection.

Vnitrni lekarstvi·2023
See all related articles

Area of Science:

  • Hematology
  • Pathophysiology

Context:

  • Hypercoagulability involves abnormal blood clot formation, increasing the risk of thrombosis.
  • This review examines inherited and acquired risk factors for hypercoagulable states.

Purpose:

  • To analyze the primary inherited and acquired causes of hypercoagulability.

Summary:

  • Inherited causes include deficiencies in natural anticoagulation proteins (antithrombin III, protein C, protein S), dysfibrinogenemia, impaired fibrinolysis, and homocystinuria.
  • Acquired causes encompass anticardiolipin antibodies, pregnancy, oral contraceptives, malignancy, nephrotic syndrome, surgery, and diabetes mellitus.

Impact:

  • Provides a comprehensive overview of hypercoagulability causes.
  • Aids in identifying individuals at risk for thrombotic events.
  • Informs clinical diagnosis and management strategies for hypercoagulable states.