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Fetal echogenic gut: a marker of intrauterine gut ischaemia?

A K Ewer1, J M McHugo, S Chapman

  • 1Birmingham Maternity Hospital.

Insights

Fetal echogenic gut (FEG) in fetuses with intrauterine growth retardation (IUGR) is associated with significant gastrointestinal feeding problems after birth. Infants with FEG experienced delayed feeding tolerance and required more interventions compared to controls.

Area of Science:

  • Perinatology
  • Neonatology
  • Pediatric Gastroenterology

Background:

  • The clinical significance of fetal echogenic gut (FEG) remains largely unknown.
  • Intrauterine growth retardation (IUGR) with absent umbilical artery end-diastolic flow velocities is a high-risk condition.
  • FEG may indicate underlying fetal gastrointestinal dysfunction.

Purpose of the Study:

  • To prospectively evaluate the gastrointestinal (GI) function in infants with FEG and IUGR.
  • To compare feeding outcomes in infants with and without FEG.

Main Methods:

  • A prospective study of nine infants with FEG and IUGR and absent umbilical artery end-diastolic flow velocities.
  • A control group of nine infants with IUGR and absent umbilical artery end-diastolic flow velocities but without FEG.
  • Postnatal evaluation of gastrointestinal function, including feeding tolerance and need for interventions.

Main Results:

  • All infants in the FEG group developed significant abdominal distension, large bile-stained aspirates, and constipation requiring interventions.
  • Enteral feeding was discontinued in the FEG group due to feeding intolerance.
  • Median time to tolerate full enteral feeds was significantly longer in the FEG group (15 days) compared to controls (4 days).
  • A higher proportion of infants in the FEG group required parenteral nutrition.

Conclusions:

  • FEG in fetuses with IUGR and absent umbilical artery end-diastolic flow velocities is predictive of significant postnatal gastrointestinal feeding problems.
  • Anticipation of feeding difficulties is crucial when FEG is detected prenatally.
  • Further research into the pathophysiology of FEG is warranted.

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