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Permeation of human chorioamniotic membranes by Escherichia coli in vitro

T N Gyr1, A Malek, F Mathez-Loic

  • 1Department of Obstetrics and Gynecology, University of Bern, Switzerland.

Abstract

Insights

Escherichia coli (E. coli) can pass through human chorioamniotic membranes in laboratory settings. These membranes offer a weak defense against ascending infections, allowing bacterial growth.

Area of Science:

  • Obstetrics and Gynecology
  • Infectious Diseases
  • Microbiology

Background:

  • Ascending infections pose a significant risk during pregnancy.
  • The chorioamniotic membranes act as a protective barrier between the maternal and fetal environments.
  • Understanding the integrity of this barrier against microbial invasion is crucial for preventing adverse pregnancy outcomes.

Purpose of the Study:

  • To investigate the in vitro permeation of Escherichia coli (E. coli) through human chorioamniotic membranes.
  • To assess the role of these membranes as a barrier against bacterial translocation.
  • To evaluate the impact of E. coli presence on membrane viability and bacterial growth.

Main Methods:

  • Human chorioamniotic membranes from term cesarean sections were used in a two-compartment in vitro model.
  • One compartment was inoculated with E. coli, and bacterial growth was monitored over 12 hours.
  • Histopathological examination confirmed bacterial permeation, and metabolic changes (glucose, lactate) were analyzed.

Main Results:

  • E. coli concentrations increased significantly in the inoculated compartment, reaching 10(10) colony-forming units/mL.
  • E. coli permeated the membranes, with detectable growth (10(3) colony-forming units/mL) in the amnion-facing compartment after 6 hours.
  • Histopathology confirmed E. coli passage, and metabolic markers showed linear changes, indicating membrane activity.

Conclusions:

  • Viable human chorioamniotic membranes are permeable to E. coli.
  • The chorioamniotic membranes provide a weak barrier against ascending infections.
  • These membranes do not inhibit bacterial growth, highlighting a potential pathway for intrauterine infections.

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