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Interferon-gamma in starch microparticles: nitric oxide-generating activity in vitro and antileishmanial effect in

L Degling1, P Stjärnkvist, I Sjöholm

  • 1Department of Pharmacy, Pharmaceutics, Uppsala University, Sweden.

Insights

Recombinant mouse interferon-gamma (mu IFN-gamma) bound to microparticles effectively activates macrophages and combats Leishmania donovani infections in mice, even at low doses ineffective in its free form.

Area of Science:

  • Immunology
  • Parasitology
  • Biomaterials Science

Background:

  • Recombinant mouse interferon-gamma (mu IFN-gamma) is a key cytokine involved in immune responses.
  • Lysosomotropic drug carriers, such as polyacryl starch microparticles, can enhance drug delivery and efficacy.
  • Leishmania donovani is a parasite responsible for visceral leishmaniasis, a serious infectious disease.

Purpose of the Study:

  • To investigate the efficacy of mu IFN-gamma covalently coupled to polyacryl starch microparticles as a therapeutic agent.
  • To evaluate the anti-leishmanial activity of microparticle-bound mu IFN-gamma in vivo.
  • To explore the mechanism of action of microparticle-bound mu IFN-gamma in activating macrophages.

Main Methods:

  • Covalent coupling of mu IFN-gamma to polyacryl starch microparticles.
  • Assessment of macrophage activation via nitrite production in vitro.
  • Evaluation of anti-leishmanial effects in Leishmania donovani-infected mice.
  • Investigation of the role of cell membrane receptors in mu IFN-gamma-mediated macrophage activation.

Main Results:

  • Microparticle-bound mu IFN-gamma activated cultured macrophages to produce nitrite.
  • Microparticle-bound mu IFN-gamma demonstrated a significant anti-leishmanial effect in infected mice.
  • Low doses of mu IFN-gamma, ineffective in free form, showed efficacy when bound to microparticles.
  • Activation of macrophages by microparticle-bound mu IFN-gamma required intact cell membrane receptors.

Conclusions:

  • Polyacryl starch microparticles serve as an effective carrier for mu IFN-gamma, enhancing its therapeutic potential.
  • Microparticle-bound mu IFN-gamma offers a promising strategy for treating Leishmania donovani infections.
  • The mechanism of action involves macrophage activation dependent on cell surface receptors.

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