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An audit of outcome of treatment in acromegaly

A S Bates1, W Van't Hoff, J M Jones

  • 1Department of Diabetes and Endocrinology, North Staffordshire Royal Infirmary, Hartshill, Stoke-on-Trent, Staffordshire.

Insights

Acromegaly continues to be linked with higher mortality rates. However, normalizing growth hormone levels significantly improves survival, suggesting a key therapeutic target for this condition.

Area of Science:

  • Endocrinology
  • Clinical Medicine
  • Epidemiology

Background:

  • Acromegaly, a disorder caused by excess growth hormone, has historically been associated with increased mortality.
  • The impact of modern treatments on acromegaly-related mortality requires ongoing investigation.

Purpose of the Study:

  • To determine if acromegaly is still associated with increased mortality.
  • To assess the influence of growth hormone levels, diabetes, and hypertension on long-term outcomes.
  • To evaluate the effectiveness of achieving specific growth hormone level targets on patient survival.

Main Methods:

  • A retrospective hospital case note review of 79 acromegaly patients followed since 1967.
  • Life table analysis was used to compare observed mortality to the general population.
  • Growth hormone secretion was assessed and compared between deceased and living patients.

Main Results:

  • Acromegaly remains associated with increased mortality (observed/expected deaths ratio = 2.68).
  • Patients with growth hormone levels below 5 mU/l exhibited survival rates comparable to the general population.
  • Higher pre-treatment growth hormone levels were observed in deceased patients; diabetes and hypertension did not significantly influence mortality.
  • Vascular causes accounted for 57% of deaths.

Conclusions:

  • Growth hormone hypersecretion is a significant contributor to excess mortality in acromegaly.
  • Therapeutic goals should focus on reducing average daytime growth hormone levels to below 5 mU/l.
  • Further large-scale studies are needed to compare treatment modalities' effects on growth hormone secretion and long-term outcomes.

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