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Expression and cloning of migration inhibitory factor-related protein (MRP)8 and MRP14 in arthritis-susceptible rats

T Imamichi1, I Uchida, S M Wahl

  • 1Cellular Immunology Section, National Institute of Dental Research, National Institutes of Health, Bethesda, MD 20892.

Insights

Rat migration inhibitory factor-related protein 8 (MRP8) and MRP14 genes are linked to chronic inflammation in LEW/N rats. Their absence in F344/N rats suggests a role in susceptibility to streptococcal cell wall-induced disease.

Area of Science:

  • Immunology
  • Molecular Biology
  • Genetics

Background:

  • Migration inhibitory factor-related proteins (MRPs) are involved in inflammatory processes.
  • Rat strains LEW/N and F344/N exhibit differential susceptibility to streptococcal cell wall (SCW)-induced arthritis.
  • Understanding genetic differences is crucial for elucidating disease mechanisms.

Purpose of the Study:

  • To identify genes differentially expressed between LEW/N and F344/N rats.
  • To investigate the role of MRP8 and MRP14 in the susceptibility to SCW-induced chronic disease.

Main Methods:

  • Subtracted cDNA library screening was employed to identify differentially expressed genes.
  • Amino acid sequence homology between rat, human, and mouse MRP8 and MRP14 was analyzed.
  • Gene expression patterns were correlated with disease development in response to SCW.

Main Results:

  • Rat migration inhibitory factor-related protein 8 (MRP8) and MRP14 were identified as differentially expressed genes.
  • Rat MRP8 and MRP14 sequences show 60-80% identity to human and mouse orthologs.
  • MRP8 and MRP14 gene expression correlated with chronic inflammation in LEW/N rats but were absent in resistant F344/N rats.

Conclusions:

  • MRP8 and MRP14 expression is associated with susceptibility to SCW-induced chronic inflammatory disease in rats.
  • These findings highlight a potential role for MRP8 and MRP14 in the pathogenesis of arthritis.
  • Further research into MRP8 and MRP14 could offer insights into inflammatory disease mechanisms.

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