Biochemical polymorphisms in wild rats (Rattus norvegicus) captured in Oita city, Japan

¹Institute for Animal Experimentation, University of Tokushima School of Medicine, Japan.

The Journal of Veterinary Medical Science

|August 1, 1993

Abstract:

The investigation of biochemical markers in wild rats (Rattus norvegicus) is thought to be useful in the efficient establishment of inbred strains with a wide range of genetic variability of wild rats. In this study, 21 marker proteins were analyzed by electrophoresis in nine wild rats captured in Oita city. It was found that eleven marker samples in six animals were heterogeneous. We further found that the investigated wild rat population possessed the interesting polymorphisms of esterase-4 (ES-4) with high frequency, which are very rare in laboratory rats.

Related Experiment Videos

Biochemical polymorphisms in wild rats (Rattus norvegicus) captured in Oita city, Japan

Related Concept Videos

Critical Appraisal Toolkit (CAT) for assessing multiple types of evidence.

Impact of observing hand hygiene in practice and research: a methodological reconsideration.

Interventions to improve hand hygiene compliance in patient care.

Interventions to improve hand hygiene compliance in patient care.

Measuring handwashing performance in health service audits and research studies.

The mock isolation room: a fun way to review infection control.

Histological and proteomic characterization of hyaline material in two dogs with hyalinizing pancreatic adenocarcinoma.

Prevalence and egg surface morphology of capillariid nematodes in red foxes (Vulpes vulpes) from Hokkaido, Japan.

Activity patterns of small- and medium-sized mammals inside and outside a sanitation control zone on a pig farm.

Copper toxicosis in 3 Japanese Black cattle with chronic cholangiohepatitis.

Effects of pulsed electromagnetic therapy on the major hematological and biochemical parameters in horses.

Comparative analysis of the cardiotoxicity induced by doxorubicin and epirubicin and exploratory investigations of clinical parameters indicating the cardiotoxicity in healthy dogs.

[Autism - Phenotypic variability].

Significant differences in phenotype and age of onset in an SPG4 family caused by whole SPAST deletion combined with a hemizygous S44L modifier.

A Novel Mutation in ACTG1 as the Probable Cause of Nonsyndromic Hearing Loss in Chinese Han Population.

Rare missense variants in NECTIN1 alter local protein structure and may contribute to non-syndromic cleft lip with or without palate.

Two Novel Compound Heterozygous CDH23 Mutations Underlying Non-Syndromic Hearing Loss.

Biallelic VPS41 Variants in Autosomal Recessive Spinocerebellar Ataxia 29 Resolved by Long-Read Sequencing and RNA Analysis.

Related Experiment Videos

Biochemical polymorphisms in wild rats (Rattus norvegicus) captured in Oita city, Japan

Abstract:

Related Concept Videos

Related Articles