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Demonstration of a relation between urinary digoxin-like immunoreactive substance and cardiac performance

H S Friedman1, B Babb, I B Shim

  • 1Section of Cardiology, Brooklyn Hospital Center.

Chest
|March 1, 1993
PubMed

Insights

Urinary digoxin-like immunoreactive substance (DLIS) correlates with cardiac performance, specifically left ventricular fractional shortening. This finding suggests DLIS may act as a ligand for cardiac glycoside receptors.

Area of Science:

  • Cardiology
  • Endocrinology
  • Biochemistry

Background:

  • Cardiac glycosides play a crucial role in regulating cardiac function.
  • The existence and physiological role of endogenous digoxin-like immunoreactive substance (DLIS) remain under investigation.
  • Understanding factors influencing DLIS levels is important for cardiac research.

Purpose of the Study:

  • To investigate the relationship between urinary DLIS concentrations and cardiac performance in individuals not receiving cardiac glycosides.
  • To identify potential physiological correlates of urinary DLIS.

Main Methods:

  • A cohort study involving 34 individuals undergoing echocardiography was conducted.
  • Echocardiographic parameters of cardiac dimensions and performance were assessed.
  • Urinary DLIS, creatinine, and electrolyte concentrations were measured.

Main Results:

  • Urinary DLIS levels varied, with a mean of 0.22 +/- 0.24 ng/ml.
  • Bivariate analyses showed significant correlations between urinary DLIS and body weight, left ventricular end-diastolic dimension, left ventricular fractional shortening, and mitral inflow velocity.
  • Multivariate analysis identified a significant independent association between urinary DLIS and left ventricular fractional shortening.

Conclusions:

  • Urinary DLIS is related to cardiac performance under steady-state conditions.
  • The findings suggest that DLIS may function as an endogenous ligand for cardiac glycoside receptors.
  • Further research is warranted to elucidate the precise physiological role of DLIS in cardiovascular regulation.
Abstract

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