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Evidence for binding of extrachromosomal DNA sequences to nuclear matrix proteins in multidrug-resistant KB-V1 cells

F Thiebaut1, A R Hanauske, D D Von Hoff

  • 1University of California San Diego Cancer Center 92103.

FEBS Letters
|March 15, 1993
PubMed

Insights

Multidrug-resistant cells contain extrachromosomal DNA (episomes) that bind to nuclear matrix proteins. This binding, reversed by proteinase K, suggests nuclear matrix regulation of episomal gene expression.

Area of Science:

  • Molecular Biology
  • Cell Biology
  • Genetics

Background:

  • Multidrug-resistant KB-V1 cells possess amplified mdr1 gene sequences on episomes.
  • The role of nuclear matrix proteins in regulating episomal gene expression is not well understood due to the absence of centromeric and telomeric sequences on episomes.

Purpose of the Study:

  • To investigate the binding of episomal DNA to nuclear matrix proteins.
  • To determine if nuclear matrix proteins can regulate episomal gene expression.

Main Methods:

  • High salt treatments, in situ hybridization, and dot blot analyses were employed to detect episomal DNA binding to nuclear matrix proteins.
  • Contour-clamped homogeneous electric field (CHEF) electrophoresis was used to assess the reversibility of this binding after enzymatic treatments.

Main Results:

  • Evidence of direct binding between episomal DNA and nuclear matrix proteins was observed.
  • This binding was found to be reversible only after incubation with trypsin or proteinase K, indicating a protein-mediated interaction.

Conclusions:

  • Circular extrachromosomal DNA (episomes) can directly bind to nuclear matrix proteins.
  • This interaction suggests that episomes may be functionally regulated by nuclear matrix proteins, similar to chromosomal DNA.

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