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A Murine Model of Subarachnoid Hemorrhage
Published on: November 21, 2013
Hyponatremia after aneurysmal subarachnoid hemorrhage
1School of Nursing, Memorial University of Newfoundland, St. John's, Canada.
Hyponatremia frequently complicates the clinical course for patients with aneurysmal subarachnoid hemorrhage (SAH). Although commonly attributed to the syndrome of inappropriate antidiuretic hormone (SIADH), emerging evidence suggests a range of pathological conditions may produce the abnormality. Effective management requires an approach to data collection which avoids suppositions about etiology, as incorrect assumptions may result in selection of interventions leading to further clinical deterioration. Adherence to comprehensive monitoring and management protocols ensures rapid detection and efficient management of this common problem after SAH.
Hyponatremia frequently complicates the clinical course for patients with aneurysmal subarachnoid hemorrhage (SAH). Although commonly attributed to the syndrome of inappropriate antidiuretic hormone (SIADH), emerging evidence suggests a range of pathological conditions may produce the abnormality. Effective management requires an approach to data collection which avoids suppositions about etiology, as incorrect assumptions may result in selection of interventions leading to further clinical deterioration. Adherence to comprehensive monitoring and management protocols ensures rapid detection and efficient management of this common problem after SAH.
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