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Inhaled nitric oxide for hemodynamic support after postpneumonectomy ARDS
J D Chiche1, J L Canivet, P Damas
1Department of Anesthesia and Intensive Care Medicine, CHU Liège, Belgium.
Intensive Care Medicine
|August 1, 1995
Summary
Inhaled nitric oxide (NO) improved oxygenation and hemodynamics in a patient with postpneumonectomy acute respiratory distress syndrome (ARDS) and pulmonary hypertension (PHT). The patient required sustained NO therapy but ultimately succumbed to the underlying disease.
Area of Science:
- Critical Care Medicine
- Pulmonary Medicine
- Cardiovascular Medicine
Background:
- Inhaled nitric oxide (NO) is known to cause pulmonary vasodilation and improve oxygenation in acute respiratory distress syndrome (ARDS) with pulmonary hypertension (PHT).
- The long-term clinical significance and potential toxicity of inhaled NO remain under investigation.
- Severe pulmonary hypertension (PHT) and right ventricular failure can complicate ARDS, particularly post-pneumonectomy.