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Related Experiment Videos

Parietal kinetic ataxia without proprioceptive deficit

J Ghika1, J Bogousslavsky, A Uske

  • 1Service de Neurologie, CHUV, Lausanne, Switzerland.

Journal of Neurology, Neurosurgery, and Psychiatry
|November 1, 1995
PubMed
Summary

Posterior parietal lesions can mimic cerebellar ataxia. This case highlights how damage to specific parietal areas, not typically associated with ataxia, can cause symptoms resembling cerebellar dysfunction.

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Area of Science:

  • Neuroscience
  • Neurology

Background:

  • Cerebellar ataxia is typically associated with dysfunction of the cerebellum.
  • The paracentral lobule is often implicated in parietal ataxia.

Observation:

  • A patient presented with acute onset "classic" cerebellar ataxia of the right arm.
  • The patient had no clinically detectable deep sensory loss.
  • Specific symptoms like wild swaying or worsening with visual suppression were absent.

Findings:

  • An acute posterior parietal infarct was identified as the cause.
  • The infarct involved areas 5 and 7, the angular gyrus, and parts of the parieto-occipital and temporal gyri.
  • The paracentral lobule remained unaffected.

Implications:

Related Experiment Videos

  • Posterior parietal lesions can present as cerebellar ataxia.
  • This may occur through disruption of projections to the ventrolateral thalamic nuclei.
  • The superior parietal gyrus may be crucial in mediating such ataxic symptoms.