Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Experiment Videos

Radiation-induced vascular damage

H Kritz1, M R Ulm, G Karanikas

  • 1Wilhelm Auerswald Atherosclerosis Research Group, University of Vienna, Austria.

VASA. Zeitschrift Fur Gefasskrankheiten
|January 1, 1995
PubMed
Summary
This summary is machine-generated.

Related Concept Videos

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

32nd International Austrian Winter Symposium : Zell am See, the Netherlands. 20-23 January 2016.

EJNMMI research·2016
Same author

[Protection of the unborn child in diagnostic and interventional radiological procedures].

Der Radiologe·2015
Same author

Impact of electroconvulsive therapy on 5-HT1A receptor binding in major depression.

Molecular psychiatry·2012
Same author

Radiosynthesis of [11C]SNAP-7941--the first PET-tracer for the melanin concentrating hormone receptor 1 (MCHR1).

Applied radiation and isotopes : including data, instrumentation and methods for use in agriculture, industry and medicine·2012
Same author

Global decrease of serotonin-1A receptor binding after electroconvulsive therapy in major depression measured by PET.

Molecular psychiatry·2012
Same author

Assessment of left ventricular volumes, ejection fraction and mass. Comparison of model-based analysis of ECG-gated (⁹⁹m)Tc-SPECT and ¹⁸F-FDG-PET.

Nuklearmedizin. Nuclear medicine·2010
Same journal

A new era in carotid prevention - critical appraisal of the CREST-2 results.

VASA. Zeitschrift fur Gefasskrankheiten·2026
Same journal

Management of venous thromboembolism in France - a nationwide clinical practice survey among vascular medicine specialists.

VASA. Zeitschrift fur Gefasskrankheiten·2026
Same journal

Endovascular salvage of distal infrainguinal bypass anastomotic stenosis following thrombectomy using a heparin-bonded covered stent.

VASA. Zeitschrift fur Gefasskrankheiten·2026
Same journal

Optimal ablation length for effective treatment of great saphenous vein insufficiency.

VASA. Zeitschrift fur Gefasskrankheiten·2026
Same journal

Long-term outcomes of covered versus bare metal stents in the reconstruction of the aortic bifurcation.

VASA. Zeitschrift fur Gefasskrankheiten·2026
Same journal

Early discharge and home treatment after acute pulmonary embolism.

VASA. Zeitschrift fur Gefasskrankheiten·2026
See all related articles

A familial plasma factor defect and elevated lipoprotein(a) were identified in a 55-year-old male. These findings, alongside localized vascular changes, suggest a potential genetic predisposition to cardiovascular disease.

Area of Science:

  • Vascular Biology
  • Genetics
  • Cardiovascular Medicine

Background:

  • Investigating the genetic and biochemical factors contributing to premature cardiovascular disease.
  • Assessing a 55-year-old male with a family history of myocardial infarction.

Observation:

  • The patient's daughter experienced myocardial infarction at 29 weeks gestation.
  • The patient had a history of testicular cancer treated with therapeutic irradiation 12 years prior.
  • The patient presented with an inborn familial plasma factor defect and elevated lipoprotein(a) [Lp(a)].

Findings:

  • Localized vascular changes were observed in the right femoral artery.
  • The patient's resting vascular system exhibited normal characteristics.
  • Biochemical analysis revealed an inborn familial plasma factor defect and increased Lp(a).

Related Experiment Videos

Implications:

  • The study highlights the potential role of inherited plasma factor defects and elevated Lp(a) in familial cardiovascular disease.
  • Understanding these factors is crucial for early diagnosis and risk stratification in affected families.
  • Further research into the interplay between genetic predisposition, Lp(a), and vascular health is warranted.