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MDR-1 expression in non-Hodgkin's lymphomas is unrelated to treatment intensity or response to therapy

M C Finnegan1, J Royds, J R Goepel

  • 1Department of Clinical Oncology, Sheffield University Medical School, England, UK.

Leukemia & Lymphoma
|July 1, 1995
PubMed

Insights

Drug resistance in non-Hodgkin's lymphomas (NHL) may not be primarily due to P-glycoprotein. MDR-1 gene expression was found in NHL tissues, but its correlation with treatment response was minimal, suggesting other resistance mechanisms.

Area of Science:

  • Oncology
  • Molecular Biology
  • Genetics

Background:

  • Over-expression of the MDR-1 gene, coding for P-glycoprotein, is a known factor in tumor drug resistance.
  • MDR-1 inducers are used in treating high-grade non-Hodgkin's lymphomas (NHL).

Purpose of the Study:

  • To investigate MDR-1 gene and P-glycoprotein expression in NHL.
  • To determine the correlation between MDR-1 expression and drug resistance in NHL patients.

Main Methods:

  • Northern blot analysis for MDR-1 mRNA expression.
  • Immunohistochemistry for P-glycoprotein localization.
  • Quantification of mRNA levels using scanning densitometry and G6PD comparison.

Main Results:

  • MDR-1 mRNA and P-glycoprotein were present in both non-malignant and NHL tissues.
  • Elevated MDR-1 mRNA levels (2-3 fold) were observed in some NHL cases compared to reactive tissues.
  • Little correlation was found between MDR-1 gene expression and treatment intensity or response.

Conclusions:

  • P-glycoprotein over-expression is likely not the primary driver of drug resistance in NHL.
  • Alternative mechanisms are probably responsible for the drug resistance observed in NHL patients.

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