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[Renal disease and trace elements]

F Marumo1, J P Li

  • 1Second Department of Internal Medicine, Tokyo Medical and Dental University.

Nihon Rinsho. Japanese Journal of Clinical Medicine
|January 1, 1996
PubMed
Summary
This summary is machine-generated.

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Trace elements impact kidney health, causing renal failure from excess intake like cadmium-induced Itai-itai disease. Chronic kidney disease patients experience trace element disturbances, such as aluminum-related issues.

Area of Science:

  • Environmental toxicology
  • Nephrology
  • Trace element metabolism

Context:

  • The relationship between trace elements and kidney disease is multifaceted.
  • Excessive intake of certain trace elements can lead to renal failure.
  • Chronic renal failure patients often exhibit altered trace element levels.

Purpose:

  • To explore the dual nature of trace element correlation with renal disease.
  • To highlight specific examples of trace element toxicity and their renal effects.
  • To discuss implications for public health and patient management.

Summary:

  • Trace element imbalances are linked to kidney disease through two main pathways: renal failure from excess intake (e.g., cadmium causing Itai-itai disease) and disturbances in chronic renal failure patients (e.g., aluminum-related dementia/bone disease).

Related Experiment Videos

  • Cadmium exposure via water pollution leads to Fanconi syndrome and osteomalacia.
  • While aluminum dementia has been mitigated by improved dialysis, fluoride in tap water poses a risk of retention and toxicity in chronic kidney disease patients.

    • Impact:

    • Informs understanding of environmental factors contributing to kidney disease.
    • Suggests a need for careful monitoring of trace elements in at-risk populations.
    • Highlights potential risks of public health interventions like water fluoridation for individuals with impaired kidney function.