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Severe hypercalcemia following neonatal liver transplantation

S M Bartosh1, S M Sprague, Y Nakagawa

  • 1Department of Pediatrics, University of Chicago Pritzker School of Medicine, Chicago, Ill, USA.

Mineral and Electrolyte Metabolism
|January 1, 1995
PubMed

Insights

A neonate receiving a liver transplant developed severe hypercalcemia due to intraoperative calcium infusions. These infusions were given to manage citrate toxicity in a baby with liver dysfunction.

Area of Science:

  • Pediatric Surgery
  • Nephrology
  • Critical Care Medicine

Background:

  • Orthotopic liver transplantation is a complex procedure for infants with liver failure.
  • Massive blood product transfusions are sometimes required during prolonged neonatal surgeries.
  • Citrate toxicity can occur during massive transfusions, especially in patients with hepatic dysfunction.

Observation:

  • A 3-week-old infant with liver failure underwent orthotopic liver transplant.
  • The infant received massive blood products during a prolonged surgical procedure.
  • Post-operatively, the infant's serum calcium level was critically high at 31.3 mg/dL.

Findings:

  • The severe hypercalcemia was a direct result of intraoperative calcium infusions.
  • Calcium infusions were administered to counteract citrate toxicity.
  • Hepatic dysfunction in the neonate impaired citrate metabolism, exacerbating the risk.

Implications:

  • This case highlights the risk of iatrogenic hypercalcemia in neonates undergoing liver transplantation.
  • Careful monitoring of serum calcium levels is crucial during and after massive transfusions in infants with hepatic dysfunction.
  • Management strategies for citrate toxicity in neonates require careful consideration of calcium balance.

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