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Haemophilia B (sixth edition): a database of point mutations and short additions and deletions

F Giannelli1, P M Green, S S Sommer

  • 1Paediatric Research Unit, London, UK.

Nucleic Acids Research
|January 1, 1996
PubMed

Insights

The updated haemophilia B database catalogs 1380 factor IX gene mutations found in patients. This resource details mutation locations, factor IX activity, and origins for better understanding of haemophilia B.

Area of Science:

  • Hematology
  • Human Genetics
  • Molecular Biology

Background:

  • Haemophilia B is a genetic bleeding disorder caused by mutations in the factor IX gene.
  • Accurate and comprehensive databases of these mutations are crucial for research and clinical management.
  • Previous editions have established the utility of this database for tracking genetic variations.

Purpose of the Study:

  • To present the sixth edition of the haemophilia B database, cataloging known factor IX mutations.
  • To provide an easily accessible, ordered compilation of genetic alterations in haemophilia B patients.
  • To include associated clinical and genetic data for each mutation.

Main Methods:

  • Compilation of published data on factor IX gene mutations from haemophilia B patients.
  • Inclusion of mutations resulting from small genetic changes (base substitutions, short insertions/deletions <30 bp).
  • Organization of 1380 patient entries by mutation nucleotide number.

Main Results:

  • The database contains 1380 patient entries detailing factor IX mutations.
  • Entries are systematically ordered by the nucleotide position of the mutation.
  • Information on factor IX activity, circulating factor IX antigen levels, and mutation origin is provided where available.

Conclusions:

  • The sixth edition offers a comprehensive and organized resource for factor IX mutations in haemophilia B.
  • This database facilitates research into genotype-phenotype correlations in haemophilia B.
  • It serves as a valuable tool for identifying mutation origins and tracking published findings.

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