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Related Experiment Videos

Late contralateral hyperhidrosis in lateral medullary infarcts

M Rousseaux1, J F Hurtevent, C Benaim

  • 1Service de Rééducation et Convalescence Neurologiques, Centre Hospitalier Universitaire, Lille, France.

Stroke
|May 1, 1996
PubMed
Summary

Contralateral hyperhidrosis, or increased sweating, can occur months after a lateral medullary infarct. Evaluating sympathetic skin response aids in understanding these sweating disorders.

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Area of Science:

  • Neurology
  • Autonomic Nervous System Research
  • Clinical Neuroscience

Background:

  • Medullary infarcts can disrupt autonomic pathways controlling bodily functions.
  • Understanding the neurological basis of sweating disorders is crucial for diagnosis and treatment.

Observation:

  • A subset of patients with lateral medullary lesions developed increased sweating on the side opposite the lesion.
  • This contralateral hyperhidrosis manifested 6-8 months post-stroke, triggered by heat, stress, and physical exertion.
  • Some patients also exhibited reduced or absent sweating on the ipsilesional side.

Findings:

  • Electrophysiological studies revealed significant asymmetry in sympathetic skin response (SSR) post-stroke.
  • SSR was notably higher on the contralateral side compared to the ipsilesional side.

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  • In some cases, SSR was absent on the side of the medullary lesion.

    • Implications:

    • Lateral medullary lesions can lead to delayed-onset contralateral hyperhidrosis.
    • The findings suggest a role for the lateral medulla in inhibiting sudomotor neurons.
    • Assessing SSR is valuable for diagnosing and explaining sweating abnormalities following medullary infarcts.