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Impairment of hippocampal mossy fiber LTD in mice lacking mGluR2

M Yokoi1, K Kobayashi, T Manabe

  • 1Department of Biological Sciences, Kyoto University Faculty of Medicine, Kyoto 606, Japan.

Science (New York, N.Y.)
|August 2, 1996
PubMed

Insights

Presynaptic metabotropic glutamate receptor 2 (mGluR2) is crucial for hippocampal long-term depression (LTD) at mossy fiber-CA3 synapses. However, this LTD is not essential for spatial learning in mice.

Area of Science:

  • Neuroscience
  • Synaptic Plasticity
  • Molecular Biology

Background:

  • Metabotropic glutamate receptor 2 (mGluR2) is localized to presynaptic terminals of hippocampal mossy fiber-CA3 synapses.
  • Understanding the precise role of presynaptic mGluR2 in synaptic function and learning is essential.

Purpose of the Study:

  • To investigate the role of presynaptic mGluR2 in synaptic plasticity at the hippocampal mossy fiber-CA3 synapses.
  • To determine the necessity of mGluR2-dependent long-term depression (LTD) for spatial learning.

Main Methods:

  • Utilized mGluR2 knockout mice.
  • Electrophysiological recordings were performed to assess basal synaptic transmission, paired-pulse facilitation, long-term potentiation (LTP), and LTD at mossy fiber-CA3 synapses.
  • Spatial learning was evaluated using water maze tasks.

Main Results:

  • mGluR2 deficiency did not affect histology, basal synaptic transmission, paired-pulse facilitation, or LTP.
  • Low-frequency stimulation-induced LTD at mossy fiber-CA3 synapses was significantly impaired in mGluR2 knockout mice.
  • Mutant mice exhibited normal performance in water maze learning tasks.

Conclusions:

  • Presynaptic mGluR2 is indispensable for the induction of LTD at hippocampal mossy fiber-CA3 synapses.
  • Hippocampal LTD mediated by mGluR2 is not required for successful spatial learning.

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