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Changes in coronary sinus pH during dipyridamole stress in patients with hypertrophic cardiomyopathy

P M Elliott1, G M Rosano, J S Gill

  • 1Department of Cardiological Sciences, St George's Hospital Medical School, London.

Insights

Chest pain in hypertrophic cardiomyopathy (HCM) is linked to myocardial ischemia. Coronary sinus pH monitoring effectively detects this ischemia, aiding in assessing its impact on patient outcomes.

Area of Science:

  • Cardiology
  • Cardiovascular Physiology

Background:

  • Angina pectoris and myocardial scarring in hypertrophic cardiomyopathy (HCM) suggest myocardial ischemia is involved in disease pathophysiology.
  • Evaluating ischemia in HCM is challenging due to frequent baseline electrocardiographic abnormalities and poor correlation of thallium-201 perfusion defects with symptoms.

Purpose of the Study:

  • To investigate the role of myocardial ischemia in patients with hypertrophic cardiomyopathy presenting with chest pain.
  • To assess the utility of coronary sinus pH monitoring as a sensitive technique for detecting myocardial ischemia in HCM.

Main Methods:

  • Studied 11 patients with HCM and chest pain, and 6 controls with atypical chest pain and normal coronary angiograms.
  • Monitored coronary sinus pH continuously during dipyridamole infusion using a catheter-mounted pH electrode.
  • Assessed baseline electrocardiography and thallium-201 scintigraphy in HCM patients.

Main Results:

  • Maximal change in coronary sinus pH during dipyridamole stress was significantly greater in HCM patients compared to controls (P=0.02).
  • In six HCM patients, chest pain development correlated with a decline in coronary sinus pH.
  • No correlation found between left ventricular dimensions, maximal wall thickness, and maximum pH change.

Conclusions:

  • The study provides evidence that chest pain in HCM patients is caused by myocardial ischemia.
  • Coronary sinus pH monitoring is a valuable method for quantifying myocardial ischemia in HCM.
  • Further research is needed to determine the role of myocardial ischemia in HCM pathophysiology and its prognostic implications.
Abstract

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