If Schizophrenia is enantiomorphism, is light the morphogen?

Schizophrenia remains an enigmatic condition. It would appear that both genetic and environmental influences are relevant to the aetiopathogenesis of the disorder. Although a great amount of research has been carried out concerning the condition, the results have often lead to further questions and attempts to more closely delineate the object of study have lead to the originally observed findings becoming tenuous. One way to objectively view some aspects of this large body of work is to consider the condition as a cerebral situs inversus, as data from various lines of reasoning have suggested an inversion of the situation seen in normal controls. The hypothesis presented here draws on existing biological evidence to argue the likely role of light as a relevant, stimulatory variable which may interact with asymmetrical cerebral maturation in the establishment of functional laterality.