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Transfer of conjugative plasmid pAM beta 1 from Lactococcus lactis to mouse intestinal bacteria

S Igimi1, C H Ryu, S H Park

  • 1Department of Biomedical Food Research, National Institute of Health, Tokyo, Japan. igimi@nih.go.jp

Insights

Plasmid pAM beta 1 can transfer from Lactococcus lactis to intestinal bacteria like Enterococcus faecalis in mice. Antibiotic pressure significantly increases this plasmid transfer within the gut environment.

Area of Science:

  • Microbiology
  • Genetics
  • Gastroenterology

Background:

  • Conjugal plasmid transfer is a key mechanism for bacterial adaptation and spread of antibiotic resistance.
  • Lactococcus lactis is a common bacterium found in fermented foods and the gut microbiome.
  • Enterococcus faecalis is a significant opportunistic pathogen often associated with hospital-acquired infections.

Purpose of the Study:

  • To investigate the in vitro and in vivo conjugal transfer of plasmid pAM beta 1 from Lactococcus lactis to intestinal bacteria in BALB/c mice.
  • To determine the frequency and conditions influencing plasmid transfer within the murine gut environment.

Main Methods:

  • Filter mating experiments were conducted for in vitro plasmid transfer analysis.
  • In vivo studies involved gastric intubation of donor Lactococcus lactis into mice, followed by analysis of fecal samples.
  • Antibiotic pressure (erythromycin) and artificial anus closure were used to modulate and assess plasmid transfer efficiency in vivo.

Main Results:

  • Plasmid pAM beta 1 transferred efficiently to Enterococcus faecalis in vitro (2.3 x 10^-3 frequency).
  • In vivo transfer was detected at low frequencies but significantly increased under erythromycin pressure.
  • Artificial anus closure in mice led to high-frequency plasmid transfer to Enterococcus faecalis (1.2 x 10^-3).

Conclusions:

  • Conjugal transfer of plasmid pAM beta 1 between Gram-positive bacteria, specifically to Enterococcus faecalis, occurs within the complex microbial environment of the mouse gut.
  • Environmental factors, such as antibiotic presence, can dramatically enhance plasmid dissemination in the gut microbiome.

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