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Dilator naris muscle

D G Mann, C T Sasaki, H Fukuda

    The Annals of Otology, Rhinology, and Laryngology
    |May 1, 1977
    PubMed
    Summary
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    The human nose, a key respiratory organ, actively controls airflow resistance via the dilator naris muscle (DNM). Its function is linked to nasal resistance levels and vagal nerve pathways.

    Area of Science:

    • Respiratory Physiology
    • Nasal Airway Mechanics

    Background:

    • The human nose significantly influences respiratory resistance through its valvular function.
    • The flow-limiting segment (FLS) at the limen nasi is crucial for this effect.
    • Active muscle contractions, specifically the dilator naris muscle (DNM), further modify nasal airflow.

    Purpose of the Study:

    • To investigate the role of the dilator naris muscle (DNM) in nasal respiratory control.
    • To quantify the relationship between ventilatory resistance and DNM activity.
    • To explore the neural pathways influencing nasal valvular function.

    Main Methods:

    • Quantitative determination of DNM behavior in humans and experimental animals.
    • Measurement of ventilatory resistance and its effect on DNM activity.

    Related Experiment Videos

  • Analysis of afferent vagal pathway involvement via pulmonary receptors.

    • Main Results:

    • Phasic DNM activity during normal breathing correlates directly with ventilatory resistance.
    • Complete elimination of resistance leads to cessation of DNM activity.
    • Prolonged reduced resistance impairs the reestablishment of dilator function.
    • DNM activity is modulated by pulmonary mechano- and pressure-receptors through vagal pathways.

    Conclusions:

    • Nasal valvular control is an active process modulated by DNM activity.
    • The integrity of vagus nerves is essential for proper nasal dilator function.
    • This study highlights the underappreciated role of nasal valvular control in respiration.