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[Detection of circulating endothelial cells: a new diagnostic test of angina at rest]

M Bory1, J Sampol, S Yvorra

  • 1Service de cardiologie A, CHU Timone, Marseille.

Archives Des Maladies Du Coeur Et Des Vaisseaux
|December 1, 1995
PubMed

Insights

Detecting circulating endothelial cells offers a new biological test for spontaneous angina, aiding retrospective diagnosis. This method shows promise for identifying plaque instability, a common trigger for angina attacks.

Area of Science:

  • Cardiology
  • Vascular Biology
  • Biomarker Discovery

Background:

  • Spontaneous angina diagnosis relies on electrocardiographic changes, lacking simple retrospective biological tests.
  • Angina attacks are often linked to atheromatous plaque instability and endothelial cell release into the bloodstream.

Purpose of the Study:

  • To evaluate the utility of detecting circulating endothelial cells (CECs) as a biological marker for spontaneous angina.
  • To assess the specificity and sensitivity of CEC detection for diagnosing spontaneous angina retrospectively.

Main Methods:

  • An immuno-magnetic method was employed to detect CECs in three patient groups admitted within 24 hours.
  • Group I: Acute myocardial infarction (n=11).
  • Group II: Spontaneous angina with ST depression and coronary stenosis (n=23).
  • Group III: Chest pain with normal angiography and negative spasm test (n=6).

Main Results:

  • CECs were detected in 100% of acute myocardial infarction patients.
  • 78% of spontaneous angina patients (Group II) showed detectable CECs.
  • Only 18% of patients with non-cardiac chest pain (Group III) had detectable CECs.
  • The test demonstrated 83% specificity and 95% positive predictive value for spontaneous angina.
  • Sensitivity and negative predictive value were 78% and 50%, respectively.

Conclusions:

  • Detection of circulating endothelial cells presents a simple, reliable test for the retrospective diagnosis of spontaneous angina.
  • The test shows high specificity and positive predictive value, though moderate sensitivity and negative predictive value suggest alternative diagnostic mechanisms in some cases.
  • Further research may explore other causes for CEC release in spontaneous angina beyond atheromatous plaque fissuration.

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