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Published on: September 28, 2022
Innominate artery compression of the trachea in infants
S Mahboubi1, M P Harty, A M Hubbard
1Department of Radiology, Children's Hospital of Philadelphia, PA 19104, USA.
Insights
Innominate artery compression may cause infant stridor, but MRI suggests intrinsic tracheal issues are the primary cause. Further investigation into tracheal abnormalities is recommended for respiratory symptoms.
Area of Science:
- Pediatric Pulmonology
- Medical Imaging
- Congenital Airway Abnormalities
Background:
- Innominate artery compression is a debated cause of infant stridor and apnea.
- Magnetic resonance imaging (MRI) is used to investigate airway issues.
- Segmental tracheomalacia is a potential contributing factor.
Purpose of the Study:
- To investigate the association between innominate artery compression and stridor in infants.
- To document airway abnormalities using MRI.
- To determine the primary cause of respiratory symptoms in infants.
Main Methods:
- Studied 40 infants presenting with stridor.
- Utilized magnetic resonance imaging (MRI) to assess airway anatomy.
- Analyzed tracheal morphology in relation to innominate artery compression.
Main Results:
- Seven out of 40 infants (17.5%) showed innominate artery compression of the trachea.
- Tracheal flattening extended beyond the point of compression.
- Two infants underwent surgical intervention, while five were managed conservatively.
Conclusions:
- Intrinsic abnormalities of the trachea appear to be the primary cause of respiratory symptoms in infants.
- While innominate artery compression may be present, it might not be the sole or main etiological factor.
- Further research into tracheal structural abnormalities is warranted for effective management of infant stridor.
Abstract:
Compression of the airway by the innominate artery has been a controversial cause of stridor and apnea in infants. Magnetic resonance imaging has been applied to document the possible association of segmental tracheomalacia. Forty infants with symptoms of stridor were studied, 7 of 40 infants aged 3-21 months (mean 10 months) had innominate artery compression on the trachea. Five infants were treated conservatively and two infants underwent surgery. Flattening of the trachea was longer than the mass compression point by the innominate artery. Therefore, intrinsic abnormality of the trachea is the basis of the respiratory symptom.
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