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Nitrite/nitrate (NOx) and sFas antigen levels in patients with multiple organ failure

S Endo1, K Inada, T Takakuwa

  • 1Critical Care and Emergency Center, Iwate Medical University, Morioka, Japan.

Research Communications in Molecular Pathology and Pharmacology
|May 1, 1996
PubMed

Insights

Elevated nitrite/nitrate (NOx) and soluble Fas antigen (sFas) levels are linked to multiple organ failure (MOF). These biomarkers decreased as MOF resolved, suggesting their role in MOF development.

Area of Science:

  • Biochemistry
  • Pathophysiology
  • Critical Care Medicine

Background:

  • Multiple organ failure (MOF) is a critical condition with high mortality.
  • The underlying mechanisms of MOF are not fully understood.
  • Nitrite/nitrate (NOx) and soluble Fas antigen (sFas) are potential biomarkers in critical illness.

Purpose of the Study:

  • To investigate the plasma levels of NOx and sFas in patients with MOF.
  • To explore the relationship between NOx, sFas, and the presence of sepsis in MOF patients.
  • To determine if NOx and sFas levels correlate with MOF severity and resolution.

Main Methods:

  • Plasma samples were collected from patients diagnosed with MOF.
  • Nitrite/nitrate (NOx) levels were measured.
  • Soluble Fas antigen (sFas) levels were quantified.
  • Statistical analyses were performed to compare levels between patient groups and assess correlations.

Main Results:

  • MOF patients exhibited elevated plasma NOx levels, particularly those with sepsis.
  • While not statistically significant, NOx levels tended to be higher in MOF patients with sepsis compared to those without infection.
  • A significant positive correlation was observed between plasma NOx and sFas levels in MOF patients.
  • Both NOx and sFas levels were markedly high during active MOF and decreased substantially upon clinical improvement.

Conclusions:

  • Elevated plasma NOx and sFas are associated with the presence and severity of multiple organ failure.
  • These findings suggest that NOx and sFas play a contributory role in the pathophysiology of MOF.
  • Monitoring NOx and sFas may offer insights into MOF progression and treatment response.

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