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Dysregulation of the polymorphonuclear leukocyte--Candida spp. interaction in HIV-positive patients

C Wenisch1, B Parschalk, K Zedwitz-Liebenstein

  • 1Department of Infectious Diseases, University Hospital of Vienna, Austria.

AIDS (London, England)
|August 1, 1996
PubMed
Abstract

Insights

Polymorphonuclear leukocytes (PML) from advanced HIV-1 infected patients show increased phagocytosis but impaired fungal killing. This suggests non-oxidative mechanisms may be key in increased fungal infections in AIDS patients.

Area of Science:

  • Immunology
  • Infectious Diseases
  • Cell Biology

Background:

  • HIV-1 infection is associated with a higher incidence of fungal infections.
  • Dysregulation in phagocytic cell responses to fungi may contribute to increased susceptibility.

Purpose of the Study:

  • To investigate the phagocytic cell function in HIV-1 infected patients.
  • To analyze the phagocytosis, reactive oxygen species production, and candicidal activity of polymorphonuclear leukocytes (PML) in advanced HIV-1 infection.

Main Methods:

  • Flow cytometry was used to analyze PML from HIV-1 infected patients (CDC stage C3) and healthy controls.
  • Phagocytic activity was measured by uptake of labeled Candida species.
  • Intracellular reactive oxygen intermediate production and candicidal activity were assessed.

Main Results:

  • PML from AIDS patients demonstrated enhanced phagocytic activity compared to controls.
  • The ability to generate reactive oxygen products was similar between AIDS patients and controls.
  • Candicidal activity was significantly reduced in PML from AIDS patients (P < 0.05).

Conclusions:

  • Impaired non-oxidative killing mechanisms in phagocytic cells may contribute to fungal infections in advanced HIV-1 disease.
  • These findings highlight a potential mechanism for increased fungal susceptibility in immunocompromised individuals.

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