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Alcoholic myopathies
J Fernández-Solà1, J M Junyent, A Urbano-Márquez
1Muscle Research Group, Hospital Clinic, Faculty of Medicine, University of Barcelona, Spain.
Current Opinion in Neurology
|October 1, 1996
Abstract:
Ethanol consumption may produce acute or chronic changes in striated skeletal muscle in a dose-dependent manner. The physiopathological mechanisms for such changes are probably related to alterations in membrane fluidity, channels, pumps and ionic transients, depression of muscle contractility, protein synthesis and genetic problems. In patients with alcoholic myopathies, multiorgan involvement is frequent and reversibility is only partially achieved by abstinence.