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Mitochondrial DNA mutations in multiple sclerosis patients with severe optic involvement

U Mayr-Wohlfart1, C Paulus, A Henneberg

  • 1Department of Pathology, University of Ulm, Germany.

Abstract

Insights

Mitochondrial DNA (mtDNA) mutations, particularly secondary LHON mutations and specific tRNA(Thr) gene substitutions, may play a role in the development of multiple sclerosis (MS) with optic involvement.

Area of Science:

  • Neurogenetics
  • Ophthalmology
  • Mitochondrial Biology

Background:

  • Familial cases of multiple sclerosis (MS) show maternal transmission.
  • MS is occasionally associated with Leber hereditary optic neuropathy (LHON).
  • Mitochondrial DNA (mtDNA) mutations are implicated in MS etiology.

Purpose of the Study:

  • Investigate the role of mtDNA mutations in MS.
  • Determine if LHON mutations are present in MS patients with visual pathway alterations.

Main Methods:

  • PCR amplification of mtDNA segments from 100 MS patients and 100 controls.
  • Restriction enzyme analysis and DNA sequencing to identify mutations.

Main Results:

  • Primary LHON mutations were not detected in MS patients.
  • MS patients exhibited a higher frequency of secondary LHON mutations compared to controls.
  • Two specific base pair substitutions in the mt tRNA(Thr) gene were significantly more prevalent in MS patients (p = 0.00018).

Conclusions:

  • Primary LHON mutations are not characteristic of MS with optic involvement.
  • Secondary LHON mutations and mt tRNA(Thr) gene substitutions may contribute to the etiology of MS with optic involvement.

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