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Erythrocyte enzymes decomposing reactive oxygen species and gestational age

J K Candlish1, L L Tho, H W Lee

  • 1Biochemistry Department, National University of Singapore, Kent Ridge.

Early Human Development
|October 2, 1995
PubMed

Insights

Antioxidant enzyme activity in newborns changes with gestational age. Superoxide dismutase (SOD) and catalase (CAT) increase, while glutathione peroxidase (GSH-Px) decreases, with lower SOD seen in bronchopulmonary dysplasia.

Area of Science:

  • Biochemistry
  • Neonatal Physiology
  • Pediatric Medicine

Background:

  • Antioxidant enzymes, including superoxide dismutase (SOD), catalase (CAT), and glutathione peroxidase (GSH-Px), play crucial roles in cellular defense against oxidative stress.
  • Understanding the developmental trajectory of these enzymes in neonates is essential for assessing their capacity to manage oxidative challenges, particularly during the transition to extrauterine life.

Purpose of the Study:

  • To investigate the activity levels of SOD, CAT, and GSH-Px in neonatal erythrocytes across a spectrum of gestational ages.
  • To determine if enzyme activity differs in neonates with respiratory distress syndrome (RDS) or bronchopulmonary dysplasia (BPD) compared to healthy controls.

Main Methods:

  • Erythrocytes were collected from neonates with varying gestational ages.
  • Enzyme assays were performed to quantify the activity of superoxide dismutase (SOD), catalase (CAT), and glutathione peroxidase (GSH-Px).
  • Enzyme activities were compared between neonates with and without respiratory morbidities (RDS, BPD) and matched controls.

Main Results:

  • Erythrocyte SOD and CAT activities showed a positive correlation with increasing gestational age.
  • Erythrocyte GSH-Px activity exhibited a negative correlation with increasing gestational age.
  • No significant difference in enzyme activities was observed between neonates with RDS and gestational age-matched controls.
  • Neonates diagnosed with bronchopulmonary dysplasia (BPD) presented with significantly lower SOD activity compared to controls.

Conclusions:

  • Neonatal erythrocyte antioxidant enzyme activity is significantly influenced by gestational age, with SOD and CAT increasing and GSH-Px decreasing as gestation progresses.
  • While RDS does not appear to directly impact these specific enzyme levels, lower SOD activity in BPD suggests a potential role for oxidative stress and SOD deficiency in the pathogenesis of this condition.
  • These findings underscore the critical role of superoxide dismutase in neonatal adaptation to extrauterine life and highlight its potential involvement in the development of chronic lung disease.

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