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D-penicillamine-induced myositis in rheumatoid arthritis

R Chappel1, J Willems

  • 1Department of Physical Medicine and Rheumatology, Middelheim Hospital, Antwerp, Belgium.

Clinical Rheumatology
|January 1, 1996
PubMed

Insights

D-penicillamine can cause inflammatory myopathy in rheumatoid arthritis patients. Electromyography (EMG) is a valuable diagnostic tool for this condition, even when muscle enzymes are normal.

Area of Science:

  • Rheumatology
  • Neurology
  • Toxicology

Background:

  • D-penicillamine is a medication used for rheumatoid arthritis (RA).
  • Drug-induced myopathy is a known adverse effect of some medications.
  • Inflammatory myopathy can present with muscle pain and weakness.

Observation:

  • A 34-year-old female on long-term D-penicillamine therapy developed proximal muscle pain and weakness.
  • Electromyography (EMG) revealed findings consistent with inflammatory muscle disease.
  • Serum muscle enzyme levels were normal in this patient.

Findings:

  • Discontinuation of D-penicillamine and initiation of prednisolone led to rapid clinical improvement.
  • EMG proved to be a sensitive diagnostic tool for D-penicillamine-induced myopathy.
  • This case highlights the utility of EMG in diagnosing inflammatory myopathy when muscle enzymes are unremarkable.

Implications:

  • Clinicians should consider D-penicillamine-induced myopathy in RA patients presenting with unexplained muscle symptoms.
  • EMG is a crucial diagnostic modality for identifying drug-induced inflammatory myopathy, especially with normal laboratory markers.
  • Early diagnosis and management can lead to favorable outcomes in patients with D-penicillamine-induced myopathy.

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