Related Experiment Videos
Cricoarytenoid joint mobility after chronic vocal cord paralysis
The Laryngoscope
|December 1, 1996
Summary
Long-standing laryngeal paralysis in humans and dogs did not show cricoarytenoid (CA) joint ankylosis. This finding supports the effectiveness of thyroplasty medialization procedures for treating vocal cord paralysis.
Area of Science:
- Laryngology
- Pathology
- Surgical Anatomy
Background:
- Vocal cord paralysis can result from damage to the recurrent laryngeal nerve.
- Long-term effects on the cricoarytenoid joint and laryngeal muscles are not fully understood.
- Thyroplasty medialization is a surgical option for vocal cord paralysis.
Purpose of the Study:
- To histopathologically examine laryngeal specimens from cases of long-standing paralysis.
- To assess for changes in the cricoarytenoid (CA) joints and intrinsic laryngeal musculature.
- To determine the presence or absence of CA joint ankylosis.
Main Methods:
- Histopathological analysis of eleven whole organ laryngeal specimens (10 human, 1 dog).
- Specimens had a history of unilateral or bilateral paralysis lasting 6 months to 17 years.
- Focus on evaluating the cricoarytenoid joints and intrinsic laryngeal muscles.
Main Results:
- No evidence of cricoarytenoid (CA) joint ankylosis was observed in any specimen.
- Ankylosis, defined as fibrous/osseous obliteration or articular surface degeneration, was absent.
- Intrinsic laryngeal musculature was not detailed in the provided abstract, but CA joint integrity was confirmed.
Conclusions:
- The absence of cricoarytenoid (CA) joint ankylosis in long-standing paralysis is a significant finding.
- This supports the potential efficacy and safety of thyroplasty medialization procedures.
- Further research may explore muscular changes in conjunction with joint integrity.