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Carotid sinus pressure: a new application of an old maneuver. A preliminary report
1Department of Intensive Care, Central Clinical Hospital, Sofia, Bulgaria.
Insights
Carotid sinus pressure (CSP) effectively distinguishes aberrant ventricular complexes from ventricular ectopy in atrial fibrillation. This simple bedside technique accurately identifies the origin of wide QRS-complexes.
Area of Science:
- Cardiology
- Electrophysiology
Background:
- Atrial fibrillation frequently presents with wide QRS-complexes.
- Distinguishing aberrant ventricular conduction from ventricular ectopy is clinically important.
Purpose of the Study:
- To evaluate the efficacy of carotid sinus pressure (CSP) in differentiating aberrant ventricular complexes from ventricular ectopy in atrial fibrillation patients.
Main Methods:
- Carotid sinus pressure (CSP) was applied during ECG monitoring in nine patients with atrial fibrillation and frequent wide QRS-complexes.
- The response to CSP was assessed by changes in ventricular rate and wide QRS-complexes.
- Adenosine and/or lidocaine boluses were used to verify the origin of wide QRS-complexes.
Main Results:
- CSP successfully resolved aberrant beats in 85.7% of patients (6/7).
- The technique demonstrated high specificity (100%), accuracy (88.9%), and predictive value (100%).
Conclusions:
- Carotid sinus pressure (CSP) is a reliable and simple bedside method for distinguishing aberrant ventricular complexes from ventricular ectopy in atrial fibrillation.
- This technique aids in clarifying the origin of wide QRS-complexes.
Abstract:
This study reveals the capability of carotid sinus pressure (CSP) in distinguishing aberrant ventricular complexes from ventricular ectopy in atrial fibrillation. Nine patients with atrial fibrillation and frequent (> 10/min) single and/or repetitive wide QRS-complexes (duration > 0.12 s) participated. None of them were on antiarrhythmic medication, included digitalis. CSP was performed during ECG monitoring and the response to it was arbitrarily designated positive if it resulted in slowing of the ventricular rate and a temporary abolition of wide QRS-complexes. The origin of wide QRS-complexes was verified by an i.v. bolus of adenosine and/or lidocaine. Our results showed that CSP resolved aberrant beats in 85.7% (six of seven patients) and had high specificity (100%), accuracy (88.9%) and predictive value (100%). In conclusion, CSP can be used as a simple bedside technique to distinguish the origin of wide QRS-complexes.
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