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Growth after bone marrow transplantation in young children conditioned with chemotherapy alone

L Adan1, M L de Lanversin, C Thalassinos

  • 1Pediatric Endocrinology, Université Paris V et Hôpital Necker- Enfants Malades, Assistance Publique-Hôpitaux de Paris, France.

Insights

Children undergoing bone marrow transplantation (BMT) may experience short stature. Post-BMT complications, not chemotherapy conditioning alone, hinder catch-up growth and insulin-like growth factor I (IGFI) increase in children.

Area of Science:

  • Pediatric Endocrinology
  • Hematology
  • Oncology

Background:

  • Short stature is a known complication of bone marrow transplantation (BMT).
  • Growth impairment can result from conditioning regimens and/or post-transplant complications.
  • Understanding factors affecting growth post-BMT is crucial for pediatric patient care.

Purpose of the Study:

  • To evaluate the impact of chemotherapy conditioning and BMT complications on growth in children.
  • To determine if chemotherapy alone prevents catch-up growth after BMT.
  • To investigate the role of post-BMT complications in growth failure.

Main Methods:

  • Study included 30 children conditioned for BMT using chemotherapy (cyclophosphamide and busulfan).
  • Children were divided into two groups based on the presence (Group 2) or absence (Group 1) of serious/prolonged post-BMT complications.
  • Growth and plasma insulin-like growth factor I (IGFI) levels were assessed 2 years post-BMT.

Main Results:

  • Group 1 (no complications) showed significant catch-up growth and increased plasma IGFI levels 2 years post-BMT.
  • Group 2 (with complications) did not exhibit significant growth or increased plasma IGFI levels.
  • Initial growth retardation due to disease did not differ between groups at the time of BMT.

Conclusions:

  • Chemotherapy conditioning alone does not preclude catch-up growth in young children post-BMT.
  • Post-BMT complications are the primary cause of impaired catch-up growth.
  • BMT complications appear to inhibit the increase in plasma IGFI, thereby preventing catch-up growth.

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