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Aging lengthens TauDD in C57BL/6J, DBA/2J, and outbred SWR male mice (Mus musculus)

A R Mayeda1, J R Hofstetter, B Possidente

  • 1Department of Veterans Affairs, Richard L. Roudebush Medical Center, Indianapolis, Indiana, USA.

Insights

Aging lengthens the free-running period (TauDD) of circadian rhythms in laboratory mice. This lengthening effect, observed across multiple strains, contrasts with findings in other rodent species.

Area of Science:

  • Chronobiology
  • Animal Models
  • Gerontology

Background:

  • Circadian rhythms regulate physiological processes in mammals.
  • The free-running period (TauDD) of circadian rhythms can be affected by aging.
  • Previous studies on rodent aging and circadian rhythms have yielded mixed results.

Purpose of the Study:

  • To investigate the effect of aging on the free-running period (TauDD) of circadian rhythms in different laboratory mouse strains.
  • To compare age-related changes in TauDD across inbred and outbred mouse populations.
  • To determine if the lengthening of TauDD with age is specific to certain mouse strains or a general species characteristic.

Main Methods:

  • Monitoring wheel-running activity to assess circadian rhythms.
  • Measuring the free-running period (TauDD) in laboratory mice (Mus musculus) at different ages.
  • Utilizing two inbred strains (DBA/2J, C57BL/6J) and one outbred strain (Tac:(SW)fBR).

Main Results:

  • TauDD increased with age in all three mouse strains studied.
  • The lengthening of TauDD was observed in DBA/2J and outbred mice, similar to previous findings in C57BL/6J mice.
  • Age-related TauDD lengthening was observed at approximately 100 and 300 days for inbred strains, and 100 and 600 days for the outbred strain.

Conclusions:

  • The lengthening of the free-running period (TauDD) with age in laboratory mice is not limited to the C57BL strain.
  • This age-related lengthening of TauDD may be a general characteristic of Mus musculus.
  • Findings contrast with observations in other rodent species, suggesting species-specific aging effects on circadian rhythms.

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